Reviews Zovirax Injection


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Zovirax Injection Drug Description
ZOVIRAX®
(acyclovir sodium) for Injection
FOR INTRAVENOUS INFUSION ONLY
DRUG DESCRIPTION



What are the possible side effects of acyclovir (Zovirax)?

Get emergency medical help if you have any of these signs of an allergic reaction: hives; difficulty breathing; swelling of your face, lips, tongue, or throat.
Stop using acyclovir and call your doctor at once if you have any of these serious side

urinating less than usual or not at all;
fever, chills, body aches, flu symptoms;
a red, blistering, peeling skin rash;
jaundice (yellowing of the skin or eyes);
swelling, pain, tenderness, or skin...
Read All Potential Side Effects for Zovirax Injection »




ZOVIRAX is the brand name for acyclovir, a synthetic nucleoside analog active
against herpesviruses. Acyclovir sodium for injection is a sterile lyophilized
powder for intravenous administration only. Each 500-mg vial contains 500 mg
of acyclovir and 49 mg of sodium, and each 1,000-mg vial contains 1,000 mg acyclovir
and 98 mg of sodium. Reconstitution of the 500-mg or 1,000-mg vials with 10
mL or 20 mL, respectively, of Sterile Water for Injection, USP results in a
solution containing 50 mg/mL of acyclovir. The pH of the reconstituted solution
is approximately 11. Further dilution in any appropriate intravenous solution
must be performed before infusion (see DOSAGE AND ADMINISTRATION: Method
of Preparation and Administration).
Acyclovir sodium is a white, crystalline powder with the molecular formula
C8H10N5NaO3 and a molecular weight
of 247.19. The maximum solubility in water at 25°C exceeds 100 mg/mL. At
physiologic pH, acyclovir sodium exists as the un-ionized form with a molecular
weight of 225 and a maximum solubility in water at 37°C of 2.5 mg/mL. The
pka's of acyclovir are 2.27 and 9.25.
The chemical name of acyclovir sodium is 2-amino-1,9-dihydro-9-[(2-hydroxyethoxy)methyl]-6H-purin-6-one
monosodium salt; it has the following structural formula:










VIROLOGY
Mechanism of Antiviral Action: Acyclovir is a synthetic purine
nucleoside analogue with in vitro and in vivo inhibitory activity against herpes
simplex virus types 1 (HSV-1), 2 (HSV-2), and varicella-zoster virus (VZV).

The inhibitory activity of acyclovir is highly selective due to its affinity
for the enzyme thymidine kinase (TK) encoded by HSV and VZV. This viral enzyme
converts acyclovir into acyclovir monophosphate, a nucleotide analogue. The
monophosphate is further converted into diphosphate by cellular guanylate kinase
and into triphosphate by a number of cellular enzymes. In vitro, acyclovir triphosphate
stops replication of herpes viral DNA. This is accomplished in 3 ways: 1) competitive
inhibition of viral DNA polymerase, 2) incorporation into and termination of
the growing viral DNA chain, and 3) inactivation of the viral DNA polymerase.
The greater antiviral activity of acyclovir against HSV compared to VZV is due
to its more efficient phosphorylation by the viral TK.
Antiviral Activities: The quantitative relationship between the
in vitro susceptibility of herpes viruses to antivirals and the clinical response
to therapy has not been established in humans, and virus sensitivity testing
has not been standardized. Sensitivity testing results, expressed as the concentration
of drug required to inhibit by 50% the growth of virus in cell culture (IC50),
vary greatly depending upon a number of factors. Using plaque-reduction assays,
the IC50 against herpes simplex virus isolates ranges from 0.02 to
13.5 mcg/mL for HSV-1 and from 0.01 to 9.9 mcg/mL for HSV-2. The IC50
for acyclovir against most laboratory strains and clinical isolates of VZV ranges
from 0.12 to 10.8 mcg/mL. Acyclovir also demonstrates activity against the Oka
vaccine strain of VZV with a mean IC50 of 1.35 mcg/mL.
Drug Resistance: Resistance of HSV and VZV to acyclovir can result
from qualitative and quantitative changes in the viral TK and/or DNA polymerase.
Clinical isolates of HSV and VZV with reduced susceptibility to acyclovir
have been recovered from immunocompromised patients, especially with advanced
HIV infection. While most of the acyclovir-resistant mutants isolated thus far
from such patients have been found to be TK-deficient mutants, other mutants
involving the viral TK gene (TK partial and TK altered) and DNA polymerase have
been isolated. TK-negative mutants may cause severe disease in infants and immunocompromised
adults. The possibility of viral resistance to acyclovir should be considered
in patients who show poor clinical response during therapy.Last reviewed on RxList: 9/7/2007




Zovirax Injection Drug Description
ZOVIRAX®
(acyclovir sodium) for Injection
FOR INTRAVENOUS INFUSION ONLY
DRUG DESCRIPTION



What are the possible side effects of acyclovir (Zovirax)?

Get emergency medical help if you have any of these signs of an allergic reaction: hives; difficulty breathing; swelling of your face, lips, tongue, or throat.
Stop using acyclovir and call your doctor at once if you have any of these serious side

urinating less than usual or not at all;
fever, chills, body aches, flu symptoms;
a red, blistering, peeling skin rash;
jaundice (yellowing of the skin or eyes);
swelling, pain, tenderness, or skin...
Read All Potential Side Effects for Zovirax Injection »




ZOVIRAX is the brand name for acyclovir, a synthetic nucleoside analog active
against herpesviruses. Acyclovir sodium for injection is a sterile lyophilized
powder for intravenous administration only. Each 500-mg vial contains 500 mg
of acyclovir and 49 mg of sodium, and each 1,000-mg vial contains 1,000 mg acyclovir
and 98 mg of sodium. Reconstitution of the 500-mg or 1,000-mg vials with 10
mL or 20 mL, respectively, of Sterile Water for Injection, USP results in a
solution containing 50 mg/mL of acyclovir. The pH of the reconstituted solution
is approximately 11. Further dilution in any appropriate intravenous solution
must be performed before infusion (see DOSAGE AND ADMINISTRATION: Method
of Preparation and Administration).
Acyclovir sodium is a white, crystalline powder with the molecular formula
C8H10N5NaO3 and a molecular weight
of 247.19. The maximum solubility in water at 25°C exceeds 100 mg/mL. At
physiologic pH, acyclovir sodium exists as the un-ionized form with a molecular
weight of 225 and a maximum solubility in water at 37°C of 2.5 mg/mL. The
pka's of acyclovir are 2.27 and 9.25.
The chemical name of acyclovir sodium is 2-amino-1,9-dihydro-9-[(2-hydroxyethoxy)methyl]-6H-purin-6-one
monosodium salt; it has the following structural formula:










VIROLOGY
Mechanism of Antiviral Action: Acyclovir is a synthetic purine
nucleoside analogue with in vitro and in vivo inhibitory activity against herpes
simplex virus types 1 (HSV-1), 2 (HSV-2), and varicella-zoster virus (VZV).

The inhibitory activity of acyclovir is highly selective due to its affinity
for the enzyme thymidine kinase (TK) encoded by HSV and VZV. This viral enzyme
converts acyclovir into acyclovir monophosphate, a nucleotide analogue. The
monophosphate is further converted into diphosphate by cellular guanylate kinase
and into triphosphate by a number of cellular enzymes. In vitro, acyclovir triphosphate
stops replication of herpes viral DNA. This is accomplished in 3 ways: 1) competitive
inhibition of viral DNA polymerase, 2) incorporation into and termination of
the growing viral DNA chain, and 3) inactivation of the viral DNA polymerase.
The greater antiviral activity of acyclovir against HSV compared to VZV is due
to its more efficient phosphorylation by the viral TK.
Antiviral Activities: The quantitative relationship between the
in vitro susceptibility of herpes viruses to antivirals and the clinical response
to therapy has not been established in humans, and virus sensitivity testing
has not been standardized. Sensitivity testing results, expressed as the concentration
of drug required to inhibit by 50% the growth of virus in cell culture (IC50),
vary greatly depending upon a number of factors. Using plaque-reduction assays,
the IC50 against herpes simplex virus isolates ranges from 0.02 to
13.5 mcg/mL for HSV-1 and from 0.01 to 9.9 mcg/mL for HSV-2. The IC50
for acyclovir against most laboratory strains and clinical isolates of VZV ranges
from 0.12 to 10.8 mcg/mL. Acyclovir also demonstrates activity against the Oka
vaccine strain of VZV with a mean IC50 of 1.35 mcg/mL.
Drug Resistance: Resistance of HSV and VZV to acyclovir can result
from qualitative and quantitative changes in the viral TK and/or DNA polymerase.
Clinical isolates of HSV and VZV with reduced susceptibility to acyclovir
have been recovered from immunocompromised patients, especially with advanced
HIV infection. While most of the acyclovir-resistant mutants isolated thus far
from such patients have been found to be TK-deficient mutants, other mutants
involving the viral TK gene (TK partial and TK altered) and DNA polymerase have
been isolated. TK-negative mutants may cause severe disease in infants and immunocompromised
adults. The possibility of viral resistance to acyclovir should be considered
in patients who show poor clinical response during therapy.Last reviewed on RxList: 9/7/2007




Zovirax Injection Drug Description
ZOVIRAX®
(acyclovir sodium) for Injection
FOR INTRAVENOUS INFUSION ONLY
DRUG DESCRIPTION



What are the possible side effects of acyclovir (Zovirax)?

Get emergency medical help if you have any of these signs of an allergic reaction: hives; difficulty breathing; swelling of your face, lips, tongue, or throat.
Stop using acyclovir and call your doctor at once if you have any of these serious side

urinating less than usual or not at all;
fever, chills, body aches, flu symptoms;
a red, blistering, peeling skin rash;
jaundice (yellowing of the skin or eyes);
swelling, pain, tenderness, or skin...
Read All Potential Side Effects for Zovirax Injection »




ZOVIRAX is the brand name for acyclovir, a synthetic nucleoside analog active
against herpesviruses. Acyclovir sodium for injection is a sterile lyophilized
powder for intravenous administration only. Each 500-mg vial contains 500 mg
of acyclovir and 49 mg of sodium, and each 1,000-mg vial contains 1,000 mg acyclovir
and 98 mg of sodium. Reconstitution of the 500-mg or 1,000-mg vials with 10
mL or 20 mL, respectively, of Sterile Water for Injection, USP results in a
solution containing 50 mg/mL of acyclovir. The pH of the reconstituted solution
is approximately 11. Further dilution in any appropriate intravenous solution
must be performed before infusion (see DOSAGE AND ADMINISTRATION: Method
of Preparation and Administration).
Acyclovir sodium is a white, crystalline powder with the molecular formula
C8H10N5NaO3 and a molecular weight
of 247.19. The maximum solubility in water at 25°C exceeds 100 mg/mL. At
physiologic pH, acyclovir sodium exists as the un-ionized form with a molecular
weight of 225 and a maximum solubility in water at 37°C of 2.5 mg/mL. The
pka's of acyclovir are 2.27 and 9.25.
The chemical name of acyclovir sodium is 2-amino-1,9-dihydro-9-[(2-hydroxyethoxy)methyl]-6H-purin-6-one
monosodium salt; it has the following structural formula:










VIROLOGY
Mechanism of Antiviral Action: Acyclovir is a synthetic purine
nucleoside analogue with in vitro and in vivo inhibitory activity against herpes
simplex virus types 1 (HSV-1), 2 (HSV-2), and varicella-zoster virus (VZV).

The inhibitory activity of acyclovir is highly selective due to its affinity
for the enzyme thymidine kinase (TK) encoded by HSV and VZV. This viral enzyme
converts acyclovir into acyclovir monophosphate, a nucleotide analogue. The
monophosphate is further converted into diphosphate by cellular guanylate kinase
and into triphosphate by a number of cellular enzymes. In vitro, acyclovir triphosphate
stops replication of herpes viral DNA. This is accomplished in 3 ways: 1) competitive
inhibition of viral DNA polymerase, 2) incorporation into and termination of
the growing viral DNA chain, and 3) inactivation of the viral DNA polymerase.
The greater antiviral activity of acyclovir against HSV compared to VZV is due
to its more efficient phosphorylation by the viral TK.
Antiviral Activities: The quantitative relationship between the
in vitro susceptibility of herpes viruses to antivirals and the clinical response
to therapy has not been established in humans, and virus sensitivity testing
has not been standardized. Sensitivity testing results, expressed as the concentration
of drug required to inhibit by 50% the growth of virus in cell culture (IC50),
vary greatly depending upon a number of factors. Using plaque-reduction assays,
the IC50 against herpes simplex virus isolates ranges from 0.02 to
13.5 mcg/mL for HSV-1 and from 0.01 to 9.9 mcg/mL for HSV-2. The IC50
for acyclovir against most laboratory strains and clinical isolates of VZV ranges
from 0.12 to 10.8 mcg/mL. Acyclovir also demonstrates activity against the Oka
vaccine strain of VZV with a mean IC50 of 1.35 mcg/mL.
Drug Resistance: Resistance of HSV and VZV to acyclovir can result
from qualitative and quantitative changes in the viral TK and/or DNA polymerase.
Clinical isolates of HSV and VZV with reduced susceptibility to acyclovir
have been recovered from immunocompromised patients, especially with advanced
HIV infection. While most of the acyclovir-resistant mutants isolated thus far
from such patients have been found to be TK-deficient mutants, other mutants
involving the viral TK gene (TK partial and TK altered) and DNA polymerase have
been isolated. TK-negative mutants may cause severe disease in infants and immunocompromised
adults. The possibility of viral resistance to acyclovir should be considered
in patients who show poor clinical response during therapy.Last reviewed on RxList: 9/7/2007




Zovirax Injection Drug Description
ZOVIRAX®
(acyclovir sodium) for Injection
FOR INTRAVENOUS INFUSION ONLY
DRUG DESCRIPTION



What are the possible side effects of acyclovir (Zovirax)?

Get emergency medical help if you have any of these signs of an allergic reaction: hives; difficulty breathing; swelling of your face, lips, tongue, or throat.
Stop using acyclovir and call your doctor at once if you have any of these serious side

urinating less than usual or not at all;
fever, chills, body aches, flu symptoms;
a red, blistering, peeling skin rash;
jaundice (yellowing of the skin or eyes);
swelling, pain, tenderness, or skin...
Read All Potential Side Effects for Zovirax Injection »




ZOVIRAX is the brand name for acyclovir, a synthetic nucleoside analog active
against herpesviruses. Acyclovir sodium for injection is a sterile lyophilized
powder for intravenous administration only. Each 500-mg vial contains 500 mg
of acyclovir and 49 mg of sodium, and each 1,000-mg vial contains 1,000 mg acyclovir
and 98 mg of sodium. Reconstitution of the 500-mg or 1,000-mg vials with 10
mL or 20 mL, respectively, of Sterile Water for Injection, USP results in a
solution containing 50 mg/mL of acyclovir. The pH of the reconstituted solution
is approximately 11. Further dilution in any appropriate intravenous solution
must be performed before infusion (see DOSAGE AND ADMINISTRATION: Method
of Preparation and Administration).
Acyclovir sodium is a white, crystalline powder with the molecular formula
C8H10N5NaO3 and a molecular weight
of 247.19. The maximum solubility in water at 25°C exceeds 100 mg/mL. At
physiologic pH, acyclovir sodium exists as the un-ionized form with a molecular
weight of 225 and a maximum solubility in water at 37°C of 2.5 mg/mL. The
pka's of acyclovir are 2.27 and 9.25.
The chemical name of acyclovir sodium is 2-amino-1,9-dihydro-9-[(2-hydroxyethoxy)methyl]-6H-purin-6-one
monosodium salt; it has the following structural formula:










VIROLOGY
Mechanism of Antiviral Action: Acyclovir is a synthetic purine
nucleoside analogue with in vitro and in vivo inhibitory activity against herpes
simplex virus types 1 (HSV-1), 2 (HSV-2), and varicella-zoster virus (VZV).

The inhibitory activity of acyclovir is highly selective due to its affinity
for the enzyme thymidine kinase (TK) encoded by HSV and VZV. This viral enzyme
converts acyclovir into acyclovir monophosphate, a nucleotide analogue. The
monophosphate is further converted into diphosphate by cellular guanylate kinase
and into triphosphate by a number of cellular enzymes. In vitro, acyclovir triphosphate
stops replication of herpes viral DNA. This is accomplished in 3 ways: 1) competitive
inhibition of viral DNA polymerase, 2) incorporation into and termination of
the growing viral DNA chain, and 3) inactivation of the viral DNA polymerase.
The greater antiviral activity of acyclovir against HSV compared to VZV is due
to its more efficient phosphorylation by the viral TK.
Antiviral Activities: The quantitative relationship between the
in vitro susceptibility of herpes viruses to antivirals and the clinical response
to therapy has not been established in humans, and virus sensitivity testing
has not been standardized. Sensitivity testing results, expressed as the concentration
of drug required to inhibit by 50% the growth of virus in cell culture (IC50),
vary greatly depending upon a number of factors. Using plaque-reduction assays,
the IC50 against herpes simplex virus isolates ranges from 0.02 to
13.5 mcg/mL for HSV-1 and from 0.01 to 9.9 mcg/mL for HSV-2. The IC50
for acyclovir against most laboratory strains and clinical isolates of VZV ranges
from 0.12 to 10.8 mcg/mL. Acyclovir also demonstrates activity against the Oka
vaccine strain of VZV with a mean IC50 of 1.35 mcg/mL.
Drug Resistance: Resistance of HSV and VZV to acyclovir can result
from qualitative and quantitative changes in the viral TK and/or DNA polymerase.
Clinical isolates of HSV and VZV with reduced susceptibility to acyclovir
have been recovered from immunocompromised patients, especially with advanced
HIV infection. While most of the acyclovir-resistant mutants isolated thus far
from such patients have been found to be TK-deficient mutants, other mutants
involving the viral TK gene (TK partial and TK altered) and DNA polymerase have
been isolated. TK-negative mutants may cause severe disease in infants and immunocompromised
adults. The possibility of viral resistance to acyclovir should be considered
in patients who show poor clinical response during therapy.Last reviewed on RxList: 9/7/2007




Zovirax Injection Drug Description
ZOVIRAX®
(acyclovir sodium) for Injection
FOR INTRAVENOUS INFUSION ONLY
DRUG DESCRIPTION



What are the possible side effects of acyclovir (Zovirax)?

Get emergency medical help if you have any of these signs of an allergic reaction: hives; difficulty breathing; swelling of your face, lips, tongue, or throat.
Stop using acyclovir and call your doctor at once if you have any of these serious side

urinating less than usual or not at all;
fever, chills, body aches, flu symptoms;
a red, blistering, peeling skin rash;
jaundice (yellowing of the skin or eyes);
swelling, pain, tenderness, or skin...
Read All Potential Side Effects for Zovirax Injection »




ZOVIRAX is the brand name for acyclovir, a synthetic nucleoside analog active
against herpesviruses. Acyclovir sodium for injection is a sterile lyophilized
powder for intravenous administration only. Each 500-mg vial contains 500 mg
of acyclovir and 49 mg of sodium, and each 1,000-mg vial contains 1,000 mg acyclovir
and 98 mg of sodium. Reconstitution of the 500-mg or 1,000-mg vials with 10
mL or 20 mL, respectively, of Sterile Water for Injection, USP results in a
solution containing 50 mg/mL of acyclovir. The pH of the reconstituted solution
is approximately 11. Further dilution in any appropriate intravenous solution
must be performed before infusion (see DOSAGE AND ADMINISTRATION: Method
of Preparation and Administration).
Acyclovir sodium is a white, crystalline powder with the molecular formula
C8H10N5NaO3 and a molecular weight
of 247.19. The maximum solubility in water at 25°C exceeds 100 mg/mL. At
physiologic pH, acyclovir sodium exists as the un-ionized form with a molecular
weight of 225 and a maximum solubility in water at 37°C of 2.5 mg/mL. The
pka's of acyclovir are 2.27 and 9.25.
The chemical name of acyclovir sodium is 2-amino-1,9-dihydro-9-[(2-hydroxyethoxy)methyl]-6H-purin-6-one
monosodium salt; it has the following structural formula:










VIROLOGY
Mechanism of Antiviral Action: Acyclovir is a synthetic purine
nucleoside analogue with in vitro and in vivo inhibitory activity against herpes
simplex virus types 1 (HSV-1), 2 (HSV-2), and varicella-zoster virus (VZV).

The inhibitory activity of acyclovir is highly selective due to its affinity
for the enzyme thymidine kinase (TK) encoded by HSV and VZV. This viral enzyme
converts acyclovir into acyclovir monophosphate, a nucleotide analogue. The
monophosphate is further converted into diphosphate by cellular guanylate kinase
and into triphosphate by a number of cellular enzymes. In vitro, acyclovir triphosphate
stops replication of herpes viral DNA. This is accomplished in 3 ways: 1) competitive
inhibition of viral DNA polymerase, 2) incorporation into and termination of
the growing viral DNA chain, and 3) inactivation of the viral DNA polymerase.
The greater antiviral activity of acyclovir against HSV compared to VZV is due
to its more efficient phosphorylation by the viral TK.
Antiviral Activities: The quantitative relationship between the
in vitro susceptibility of herpes viruses to antivirals and the clinical response
to therapy has not been established in humans, and virus sensitivity testing
has not been standardized. Sensitivity testing results, expressed as the concentration
of drug required to inhibit by 50% the growth of virus in cell culture (IC50),
vary greatly depending upon a number of factors. Using plaque-reduction assays,
the IC50 against herpes simplex virus isolates ranges from 0.02 to
13.5 mcg/mL for HSV-1 and from 0.01 to 9.9 mcg/mL for HSV-2. The IC50
for acyclovir against most laboratory strains and clinical isolates of VZV ranges
from 0.12 to 10.8 mcg/mL. Acyclovir also demonstrates activity against the Oka
vaccine strain of VZV with a mean IC50 of 1.35 mcg/mL.
Drug Resistance: Resistance of HSV and VZV to acyclovir can result
from qualitative and quantitative changes in the viral TK and/or DNA polymerase.
Clinical isolates of HSV and VZV with reduced susceptibility to acyclovir
have been recovered from immunocompromised patients, especially with advanced
HIV infection. While most of the acyclovir-resistant mutants isolated thus far
from such patients have been found to be TK-deficient mutants, other mutants
involving the viral TK gene (TK partial and TK altered) and DNA polymerase have
been isolated. TK-negative mutants may cause severe disease in infants and immunocompromised
adults. The possibility of viral resistance to acyclovir should be considered
in patients who show poor clinical response during therapy.Last reviewed on RxList: 9/7/2007





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