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Kantrex Drug Description
Kantrex
(kanamycin) Injection, Solution

WARNING
Patients treated with aminoglycosides by any route should be under close clinical
observation because of the potential toxicity associated with their use. As
with other aminoglycosides, the major toxic effects of kanamycin sulfate are
its action on the auditory and vestibular branches of the eighth nerve and the
renal tubules. Neurotoxicity is manifested by bilateral auditory toxicity which
often is permanent and, sometimes, by vestibular ototoxicity. Loss of high frequency
perception usually occurs before there is noticeable clinical hearing loss and
can be detected by audiometric testing. There may not be clinical symptoms to
warn of developing cochlear damage. Vertigo may occur and may be evidence of
vestibular injury. Other manifestations of neurotoxicity may include numbness,
skin tingling, muscle twitching, and convulsions. The risk of hearing loss increases
with the degree of exposure to either high peak or high trough serum concentrations
and continues to progress after drug withdrawal.
Renal impairment may be characterized by decreased creatinine clearance, the
presence of cells or casts, oliguria, proteinuria, decreased urine specific
gravity, or evidence of increasing nitrogen retention (increasing BUN, NPN,
or serum creatinine).
The risks of severe ototoxic and nephrotoxic reactions are sharply increased
in patients with impaired renal function and in those with normal renal function
who receive high doses or prolonged therapy.
Renal and eighth nerve function should be closely monitored, especially in
patients with known or suspected reduced renal function at the onset of therapy,
and also in those whose renal function is initially normal but who develop signs
of renal dysfunction during therapy. Serum concentrations of parenterally administered
aminoglycosides should be monitored when feasible to assure adequate levels
and to avoid potentially toxic levels. Urine should be examined for decreased
specific gravity, increased excretion of protein and the presence of cells or
casts. Blood urea nitrogen, serum creatinine, or creatinine clearance should
be measured periodically. Serial audiograms should be obtained when feasible
in patients old enough to be tested, particularly high risk patients. Evidence
of ototoxicity (dizziness, vertigo, tinnitus, roaring in the ears, and hearing
loss) or nephrotoxicity requires dosage adjustment or discontinuance of the
drug. Neuromuscular blockade with respiratory paralysis may occur when kanamycin
sulfate is instilled intraperitoneally concomitantly with anesthesia and muscle-relaxing
drugs.
Neuromuscular blockade has been reported following parenteral injection and
the oral use of aminoglycosides. The possibility of the occurrence of neuromuscular
blockade and respiratory paralysis should be considered if aminoglycosides are
administered by any route, especially in patients receiving anesthetics, neuromuscular-blocking
agents such as tubocurarine, succinylcholine, decamethonium, or in patients
receiving massive transfusions of citrate-anticoagulated blood. If blockage
occurs, calcium salts may reduce these phenomena but mechanical respiratory
assistance may be necessary.
The concurrent and/or sequential systemic, oral, or topical use of kanamycin
and other potentially nephrotoxic, and/or neurotoxic drugs, particularly polymyxin
B, bacitracin, colistin, amphotericin B, cisplatin, vancomycin, and all other
aminoglycosides (including paromomycin) should be avoided because the toxicity
may be additive. Other factors which may increase patient risk of toxicity are
advanced age and dehydration.
Kanamycin sulfate should not be given concurrently with potent diuretics (ethacrynic
acid, furosemide, meralluride sodium, sodium mercaptomerin, or mannitol). Some
diuretics themselves cause ototoxicity, and intravenously administered diuretics
may enhance aminoglycoside toxicity by altering antibiotic concentrations in
serum and tissue.

DRUG DESCRIPTION



What are the possible side effects of kanamycin (Kantrex)?

Get emergency medical help if you have any of these signs of an allergic reaction: hives; difficulty breathing; swelling of your face, lips, tongue, or throat.
Stop using kanamycin and call your doctor at once if you have any of these serious side effects:

changes in your hearing;
spinning sensation, problems with balance;
ringing or roaring sound in your ears;
numbness or tingling of your skin;
muscle twitching, seizure (convulsions); or
...
Read All Potential Side Effects for Kantrex »




Kanamycin sulfate is an aminoglycoside antibiotic produced by Streptomyces
kanamyceticus. It is C18H36N4O11
• 2H2SO4.D-Streptamine, O-3-amino-3-deoxy-α-D-glucopyranosyl
• (1→6)-O- [6-amino-6-deoxy-α-D-glucopyranosyl- (1→4)]-2-deoxy,
sulfate 1:2 (salt). It consists of two amino sugars glycosidically linked to
deoxystreptamine.








Kanamycin Injection, USP, sterile solution for parenteral administration, contains
respectively; kanamycin sulfate 75 mg, 500 mg, and 1.0 g; sodium bisulfite,
an antioxidant, 0.099%, 0.66%, and 0.45%; and sodium citrate, 0.33% 2.2%, and
2.2% with pH of each dosage form adjusted to 4.5 with sulfuric acid.
Vial headspace contains nitrogen.Last reviewed on RxList: 10/8/2008




Kantrex Drug Description
Kantrex
(kanamycin) Injection, Solution

WARNING
Patients treated with aminoglycosides by any route should be under close clinical
observation because of the potential toxicity associated with their use. As
with other aminoglycosides, the major toxic effects of kanamycin sulfate are
its action on the auditory and vestibular branches of the eighth nerve and the
renal tubules. Neurotoxicity is manifested by bilateral auditory toxicity which
often is permanent and, sometimes, by vestibular ototoxicity. Loss of high frequency
perception usually occurs before there is noticeable clinical hearing loss and
can be detected by audiometric testing. There may not be clinical symptoms to
warn of developing cochlear damage. Vertigo may occur and may be evidence of
vestibular injury. Other manifestations of neurotoxicity may include numbness,
skin tingling, muscle twitching, and convulsions. The risk of hearing loss increases
with the degree of exposure to either high peak or high trough serum concentrations
and continues to progress after drug withdrawal.
Renal impairment may be characterized by decreased creatinine clearance, the
presence of cells or casts, oliguria, proteinuria, decreased urine specific
gravity, or evidence of increasing nitrogen retention (increasing BUN, NPN,
or serum creatinine).
The risks of severe ototoxic and nephrotoxic reactions are sharply increased
in patients with impaired renal function and in those with normal renal function
who receive high doses or prolonged therapy.
Renal and eighth nerve function should be closely monitored, especially in
patients with known or suspected reduced renal function at the onset of therapy,
and also in those whose renal function is initially normal but who develop signs
of renal dysfunction during therapy. Serum concentrations of parenterally administered
aminoglycosides should be monitored when feasible to assure adequate levels
and to avoid potentially toxic levels. Urine should be examined for decreased
specific gravity, increased excretion of protein and the presence of cells or
casts. Blood urea nitrogen, serum creatinine, or creatinine clearance should
be measured periodically. Serial audiograms should be obtained when feasible
in patients old enough to be tested, particularly high risk patients. Evidence
of ototoxicity (dizziness, vertigo, tinnitus, roaring in the ears, and hearing
loss) or nephrotoxicity requires dosage adjustment or discontinuance of the
drug. Neuromuscular blockade with respiratory paralysis may occur when kanamycin
sulfate is instilled intraperitoneally concomitantly with anesthesia and muscle-relaxing
drugs.
Neuromuscular blockade has been reported following parenteral injection and
the oral use of aminoglycosides. The possibility of the occurrence of neuromuscular
blockade and respiratory paralysis should be considered if aminoglycosides are
administered by any route, especially in patients receiving anesthetics, neuromuscular-blocking
agents such as tubocurarine, succinylcholine, decamethonium, or in patients
receiving massive transfusions of citrate-anticoagulated blood. If blockage
occurs, calcium salts may reduce these phenomena but mechanical respiratory
assistance may be necessary.
The concurrent and/or sequential systemic, oral, or topical use of kanamycin
and other potentially nephrotoxic, and/or neurotoxic drugs, particularly polymyxin
B, bacitracin, colistin, amphotericin B, cisplatin, vancomycin, and all other
aminoglycosides (including paromomycin) should be avoided because the toxicity
may be additive. Other factors which may increase patient risk of toxicity are
advanced age and dehydration.
Kanamycin sulfate should not be given concurrently with potent diuretics (ethacrynic
acid, furosemide, meralluride sodium, sodium mercaptomerin, or mannitol). Some
diuretics themselves cause ototoxicity, and intravenously administered diuretics
may enhance aminoglycoside toxicity by altering antibiotic concentrations in
serum and tissue.

DRUG DESCRIPTION



What are the possible side effects of kanamycin (Kantrex)?

Get emergency medical help if you have any of these signs of an allergic reaction: hives; difficulty breathing; swelling of your face, lips, tongue, or throat.
Stop using kanamycin and call your doctor at once if you have any of these serious side effects:

changes in your hearing;
spinning sensation, problems with balance;
ringing or roaring sound in your ears;
numbness or tingling of your skin;
muscle twitching, seizure (convulsions); or
...
Read All Potential Side Effects for Kantrex »




Kanamycin sulfate is an aminoglycoside antibiotic produced by Streptomyces
kanamyceticus. It is C18H36N4O11
• 2H2SO4.D-Streptamine, O-3-amino-3-deoxy-α-D-glucopyranosyl
• (1→6)-O- [6-amino-6-deoxy-α-D-glucopyranosyl- (1→4)]-2-deoxy,
sulfate 1:2 (salt). It consists of two amino sugars glycosidically linked to
deoxystreptamine.








Kanamycin Injection, USP, sterile solution for parenteral administration, contains
respectively; kanamycin sulfate 75 mg, 500 mg, and 1.0 g; sodium bisulfite,
an antioxidant, 0.099%, 0.66%, and 0.45%; and sodium citrate, 0.33% 2.2%, and
2.2% with pH of each dosage form adjusted to 4.5 with sulfuric acid.
Vial headspace contains nitrogen.Last reviewed on RxList: 10/8/2008




Kantrex Drug Description
Kantrex
(kanamycin) Injection, Solution

WARNING
Patients treated with aminoglycosides by any route should be under close clinical
observation because of the potential toxicity associated with their use. As
with other aminoglycosides, the major toxic effects of kanamycin sulfate are
its action on the auditory and vestibular branches of the eighth nerve and the
renal tubules. Neurotoxicity is manifested by bilateral auditory toxicity which
often is permanent and, sometimes, by vestibular ototoxicity. Loss of high frequency
perception usually occurs before there is noticeable clinical hearing loss and
can be detected by audiometric testing. There may not be clinical symptoms to
warn of developing cochlear damage. Vertigo may occur and may be evidence of
vestibular injury. Other manifestations of neurotoxicity may include numbness,
skin tingling, muscle twitching, and convulsions. The risk of hearing loss increases
with the degree of exposure to either high peak or high trough serum concentrations
and continues to progress after drug withdrawal.
Renal impairment may be characterized by decreased creatinine clearance, the
presence of cells or casts, oliguria, proteinuria, decreased urine specific
gravity, or evidence of increasing nitrogen retention (increasing BUN, NPN,
or serum creatinine).
The risks of severe ototoxic and nephrotoxic reactions are sharply increased
in patients with impaired renal function and in those with normal renal function
who receive high doses or prolonged therapy.
Renal and eighth nerve function should be closely monitored, especially in
patients with known or suspected reduced renal function at the onset of therapy,
and also in those whose renal function is initially normal but who develop signs
of renal dysfunction during therapy. Serum concentrations of parenterally administered
aminoglycosides should be monitored when feasible to assure adequate levels
and to avoid potentially toxic levels. Urine should be examined for decreased
specific gravity, increased excretion of protein and the presence of cells or
casts. Blood urea nitrogen, serum creatinine, or creatinine clearance should
be measured periodically. Serial audiograms should be obtained when feasible
in patients old enough to be tested, particularly high risk patients. Evidence
of ototoxicity (dizziness, vertigo, tinnitus, roaring in the ears, and hearing
loss) or nephrotoxicity requires dosage adjustment or discontinuance of the
drug. Neuromuscular blockade with respiratory paralysis may occur when kanamycin
sulfate is instilled intraperitoneally concomitantly with anesthesia and muscle-relaxing
drugs.
Neuromuscular blockade has been reported following parenteral injection and
the oral use of aminoglycosides. The possibility of the occurrence of neuromuscular
blockade and respiratory paralysis should be considered if aminoglycosides are
administered by any route, especially in patients receiving anesthetics, neuromuscular-blocking
agents such as tubocurarine, succinylcholine, decamethonium, or in patients
receiving massive transfusions of citrate-anticoagulated blood. If blockage
occurs, calcium salts may reduce these phenomena but mechanical respiratory
assistance may be necessary.
The concurrent and/or sequential systemic, oral, or topical use of kanamycin
and other potentially nephrotoxic, and/or neurotoxic drugs, particularly polymyxin
B, bacitracin, colistin, amphotericin B, cisplatin, vancomycin, and all other
aminoglycosides (including paromomycin) should be avoided because the toxicity
may be additive. Other factors which may increase patient risk of toxicity are
advanced age and dehydration.
Kanamycin sulfate should not be given concurrently with potent diuretics (ethacrynic
acid, furosemide, meralluride sodium, sodium mercaptomerin, or mannitol). Some
diuretics themselves cause ototoxicity, and intravenously administered diuretics
may enhance aminoglycoside toxicity by altering antibiotic concentrations in
serum and tissue.

DRUG DESCRIPTION



What are the possible side effects of kanamycin (Kantrex)?

Get emergency medical help if you have any of these signs of an allergic reaction: hives; difficulty breathing; swelling of your face, lips, tongue, or throat.
Stop using kanamycin and call your doctor at once if you have any of these serious side effects:

changes in your hearing;
spinning sensation, problems with balance;
ringing or roaring sound in your ears;
numbness or tingling of your skin;
muscle twitching, seizure (convulsions); or
...
Read All Potential Side Effects for Kantrex »




Kanamycin sulfate is an aminoglycoside antibiotic produced by Streptomyces
kanamyceticus. It is C18H36N4O11
• 2H2SO4.D-Streptamine, O-3-amino-3-deoxy-α-D-glucopyranosyl
• (1→6)-O- [6-amino-6-deoxy-α-D-glucopyranosyl- (1→4)]-2-deoxy,
sulfate 1:2 (salt). It consists of two amino sugars glycosidically linked to
deoxystreptamine.








Kanamycin Injection, USP, sterile solution for parenteral administration, contains
respectively; kanamycin sulfate 75 mg, 500 mg, and 1.0 g; sodium bisulfite,
an antioxidant, 0.099%, 0.66%, and 0.45%; and sodium citrate, 0.33% 2.2%, and
2.2% with pH of each dosage form adjusted to 4.5 with sulfuric acid.
Vial headspace contains nitrogen.Last reviewed on RxList: 10/8/2008




Kantrex Drug Description
Kantrex
(kanamycin) Injection, Solution

WARNING
Patients treated with aminoglycosides by any route should be under close clinical
observation because of the potential toxicity associated with their use. As
with other aminoglycosides, the major toxic effects of kanamycin sulfate are
its action on the auditory and vestibular branches of the eighth nerve and the
renal tubules. Neurotoxicity is manifested by bilateral auditory toxicity which
often is permanent and, sometimes, by vestibular ototoxicity. Loss of high frequency
perception usually occurs before there is noticeable clinical hearing loss and
can be detected by audiometric testing. There may not be clinical symptoms to
warn of developing cochlear damage. Vertigo may occur and may be evidence of
vestibular injury. Other manifestations of neurotoxicity may include numbness,
skin tingling, muscle twitching, and convulsions. The risk of hearing loss increases
with the degree of exposure to either high peak or high trough serum concentrations
and continues to progress after drug withdrawal.
Renal impairment may be characterized by decreased creatinine clearance, the
presence of cells or casts, oliguria, proteinuria, decreased urine specific
gravity, or evidence of increasing nitrogen retention (increasing BUN, NPN,
or serum creatinine).
The risks of severe ototoxic and nephrotoxic reactions are sharply increased
in patients with impaired renal function and in those with normal renal function
who receive high doses or prolonged therapy.
Renal and eighth nerve function should be closely monitored, especially in
patients with known or suspected reduced renal function at the onset of therapy,
and also in those whose renal function is initially normal but who develop signs
of renal dysfunction during therapy. Serum concentrations of parenterally administered
aminoglycosides should be monitored when feasible to assure adequate levels
and to avoid potentially toxic levels. Urine should be examined for decreased
specific gravity, increased excretion of protein and the presence of cells or
casts. Blood urea nitrogen, serum creatinine, or creatinine clearance should
be measured periodically. Serial audiograms should be obtained when feasible
in patients old enough to be tested, particularly high risk patients. Evidence
of ototoxicity (dizziness, vertigo, tinnitus, roaring in the ears, and hearing
loss) or nephrotoxicity requires dosage adjustment or discontinuance of the
drug. Neuromuscular blockade with respiratory paralysis may occur when kanamycin
sulfate is instilled intraperitoneally concomitantly with anesthesia and muscle-relaxing
drugs.
Neuromuscular blockade has been reported following parenteral injection and
the oral use of aminoglycosides. The possibility of the occurrence of neuromuscular
blockade and respiratory paralysis should be considered if aminoglycosides are
administered by any route, especially in patients receiving anesthetics, neuromuscular-blocking
agents such as tubocurarine, succinylcholine, decamethonium, or in patients
receiving massive transfusions of citrate-anticoagulated blood. If blockage
occurs, calcium salts may reduce these phenomena but mechanical respiratory
assistance may be necessary.
The concurrent and/or sequential systemic, oral, or topical use of kanamycin
and other potentially nephrotoxic, and/or neurotoxic drugs, particularly polymyxin
B, bacitracin, colistin, amphotericin B, cisplatin, vancomycin, and all other
aminoglycosides (including paromomycin) should be avoided because the toxicity
may be additive. Other factors which may increase patient risk of toxicity are
advanced age and dehydration.
Kanamycin sulfate should not be given concurrently with potent diuretics (ethacrynic
acid, furosemide, meralluride sodium, sodium mercaptomerin, or mannitol). Some
diuretics themselves cause ototoxicity, and intravenously administered diuretics
may enhance aminoglycoside toxicity by altering antibiotic concentrations in
serum and tissue.

DRUG DESCRIPTION



What are the possible side effects of kanamycin (Kantrex)?

Get emergency medical help if you have any of these signs of an allergic reaction: hives; difficulty breathing; swelling of your face, lips, tongue, or throat.
Stop using kanamycin and call your doctor at once if you have any of these serious side effects:

changes in your hearing;
spinning sensation, problems with balance;
ringing or roaring sound in your ears;
numbness or tingling of your skin;
muscle twitching, seizure (convulsions); or
...
Read All Potential Side Effects for Kantrex »




Kanamycin sulfate is an aminoglycoside antibiotic produced by Streptomyces
kanamyceticus. It is C18H36N4O11
• 2H2SO4.D-Streptamine, O-3-amino-3-deoxy-α-D-glucopyranosyl
• (1→6)-O- [6-amino-6-deoxy-α-D-glucopyranosyl- (1→4)]-2-deoxy,
sulfate 1:2 (salt). It consists of two amino sugars glycosidically linked to
deoxystreptamine.








Kanamycin Injection, USP, sterile solution for parenteral administration, contains
respectively; kanamycin sulfate 75 mg, 500 mg, and 1.0 g; sodium bisulfite,
an antioxidant, 0.099%, 0.66%, and 0.45%; and sodium citrate, 0.33% 2.2%, and
2.2% with pH of each dosage form adjusted to 4.5 with sulfuric acid.
Vial headspace contains nitrogen.Last reviewed on RxList: 10/8/2008




Kantrex Drug Description
Kantrex
(kanamycin) Injection, Solution

WARNING
Patients treated with aminoglycosides by any route should be under close clinical
observation because of the potential toxicity associated with their use. As
with other aminoglycosides, the major toxic effects of kanamycin sulfate are
its action on the auditory and vestibular branches of the eighth nerve and the
renal tubules. Neurotoxicity is manifested by bilateral auditory toxicity which
often is permanent and, sometimes, by vestibular ototoxicity. Loss of high frequency
perception usually occurs before there is noticeable clinical hearing loss and
can be detected by audiometric testing. There may not be clinical symptoms to
warn of developing cochlear damage. Vertigo may occur and may be evidence of
vestibular injury. Other manifestations of neurotoxicity may include numbness,
skin tingling, muscle twitching, and convulsions. The risk of hearing loss increases
with the degree of exposure to either high peak or high trough serum concentrations
and continues to progress after drug withdrawal.
Renal impairment may be characterized by decreased creatinine clearance, the
presence of cells or casts, oliguria, proteinuria, decreased urine specific
gravity, or evidence of increasing nitrogen retention (increasing BUN, NPN,
or serum creatinine).
The risks of severe ototoxic and nephrotoxic reactions are sharply increased
in patients with impaired renal function and in those with normal renal function
who receive high doses or prolonged therapy.
Renal and eighth nerve function should be closely monitored, especially in
patients with known or suspected reduced renal function at the onset of therapy,
and also in those whose renal function is initially normal but who develop signs
of renal dysfunction during therapy. Serum concentrations of parenterally administered
aminoglycosides should be monitored when feasible to assure adequate levels
and to avoid potentially toxic levels. Urine should be examined for decreased
specific gravity, increased excretion of protein and the presence of cells or
casts. Blood urea nitrogen, serum creatinine, or creatinine clearance should
be measured periodically. Serial audiograms should be obtained when feasible
in patients old enough to be tested, particularly high risk patients. Evidence
of ototoxicity (dizziness, vertigo, tinnitus, roaring in the ears, and hearing
loss) or nephrotoxicity requires dosage adjustment or discontinuance of the
drug. Neuromuscular blockade with respiratory paralysis may occur when kanamycin
sulfate is instilled intraperitoneally concomitantly with anesthesia and muscle-relaxing
drugs.
Neuromuscular blockade has been reported following parenteral injection and
the oral use of aminoglycosides. The possibility of the occurrence of neuromuscular
blockade and respiratory paralysis should be considered if aminoglycosides are
administered by any route, especially in patients receiving anesthetics, neuromuscular-blocking
agents such as tubocurarine, succinylcholine, decamethonium, or in patients
receiving massive transfusions of citrate-anticoagulated blood. If blockage
occurs, calcium salts may reduce these phenomena but mechanical respiratory
assistance may be necessary.
The concurrent and/or sequential systemic, oral, or topical use of kanamycin
and other potentially nephrotoxic, and/or neurotoxic drugs, particularly polymyxin
B, bacitracin, colistin, amphotericin B, cisplatin, vancomycin, and all other
aminoglycosides (including paromomycin) should be avoided because the toxicity
may be additive. Other factors which may increase patient risk of toxicity are
advanced age and dehydration.
Kanamycin sulfate should not be given concurrently with potent diuretics (ethacrynic
acid, furosemide, meralluride sodium, sodium mercaptomerin, or mannitol). Some
diuretics themselves cause ototoxicity, and intravenously administered diuretics
may enhance aminoglycoside toxicity by altering antibiotic concentrations in
serum and tissue.

DRUG DESCRIPTION



What are the possible side effects of kanamycin (Kantrex)?

Get emergency medical help if you have any of these signs of an allergic reaction: hives; difficulty breathing; swelling of your face, lips, tongue, or throat.
Stop using kanamycin and call your doctor at once if you have any of these serious side effects:

changes in your hearing;
spinning sensation, problems with balance;
ringing or roaring sound in your ears;
numbness or tingling of your skin;
muscle twitching, seizure (convulsions); or
...
Read All Potential Side Effects for Kantrex »




Kanamycin sulfate is an aminoglycoside antibiotic produced by Streptomyces
kanamyceticus. It is C18H36N4O11
• 2H2SO4.D-Streptamine, O-3-amino-3-deoxy-α-D-glucopyranosyl
• (1→6)-O- [6-amino-6-deoxy-α-D-glucopyranosyl- (1→4)]-2-deoxy,
sulfate 1:2 (salt). It consists of two amino sugars glycosidically linked to
deoxystreptamine.








Kanamycin Injection, USP, sterile solution for parenteral administration, contains
respectively; kanamycin sulfate 75 mg, 500 mg, and 1.0 g; sodium bisulfite,
an antioxidant, 0.099%, 0.66%, and 0.45%; and sodium citrate, 0.33% 2.2%, and
2.2% with pH of each dosage form adjusted to 4.5 with sulfuric acid.
Vial headspace contains nitrogen.Last reviewed on RxList: 10/8/2008





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Kanamayin could have done a sin,In west Berlin he done them in,Shuffling cards he many did win,Playing, crossing, cheating,He threw all the morals to the bin.Kanamayin, he could […]


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Other reviews about Kanamycin on web:

Kanamycin sulfate is an aminoglycoside antibiotic, available in both oral and intravenous forms, and used to treat a wide variety of infections. Kanamycin is isolated from Streptomyces ... Kanamycin - Wikipedia, the free encyclopedia


kanamycin n. A water-soluble broad-spectrum antibiotic, C 18 H 36 O 11 N 4 , obtained from the soil bacterium Streptomyces kanamyceticus kanamycin: Definition from Answers.com


Learn about the prescription medication Kantrex (Kanamycin), drug uses, dosage, side effects, drug interactions, warnings, reviews and patient labeling. Kantrex (Kanamycin) Drug Information: Uses, Side Effects, Drug ...


All about Kanamycin. View complete and up to date Kanamycin information - part of the Drugs.com trusted medication database. Kanamycin Facts and Comparisons at Drugs.com


Kanamycin Brand names: Kantrexテδづつョ Chemical formula: Drug Forms: Kanamycin Sulfate Solution for injection (below) Kanamycin injection Kanamycin oral Kanamycin: Information from Answers.com


kanamycin /kanテδづつキaテδづつキmyテδづつキcin/ (kanテδ「テつテつウah-miテδづつエsin) an aminoglycoside antibiotic derived from Streptomyces kanamyceticus, effective against aerobic gram-negative bacilli and some gram ... kanamycin - definition of kanamycin in the Medical dictionary - by ...


kanamycin patient information. Detailed drug information for the consumer, includes dosage, kanamycin side effects and more. kanamycin consumer information from Drugs.com


This information has been developed and provided by an independent third-party source. Merck & Co., Inc. does not endorse and is not responsible for the accuracy of the content, or ... Kanamycin: Drug Information Provided by Lexi-Comp: Merck Manual ...


kanamycin (テδ凝つ渓テδεつヲnテδ嘉つ凖δ凝つmaテδ嘉つェsテδ嘉つェn) テδ「テつテつ n: an aminoglycoside antibiotic obtained from the soil bacterium Streptomyces kanamyceticus, used in the treatment of various infections, esp those ... Kanamycin | Define Kanamycin at Dictionary.com


Learn more about Kanamycin. Find the Web's best health guides, medical reports, news, videos and tools for Kanamycin. Share Kanamycin experiences and get advice from experts. Kanamycin Overview - References, Advice, News, Videos, Coping ...





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Astepro Drug Description ASTEPRO (azelastine hydrochloride) Nasal Spray 0.1% ASTEPRO (azelastine hydrochloride) Nasal Spray 0.15% DRUG DESCRIPTION What are the possible side effects of azelastine nasal (Astelin, Astepro)? Get...
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